Publikationen der ALS-Forschungsgruppe an der Charité
The EAAT2 (GLT-1) gene in motor neuron disease: absence of mutations in amyotrophic lateral sclerosis and a point mutation in patients with hereditary spastic paraplegia
Meyer T, Münch C, Völkel H, Booms P, Ludolph AC. The EAAT2
Studies of the coding region of the neuronal glutamate transporter gene in amyotrophic lateral sclerosis
Meyer T, Lenk U, Küther G, Weindl A, Speer A, Ludolph AC.
The EAAT2 (GLT-1) gene in motor neuron disease: absence of mutations in amyotrophic lateral sclerosis and a point mutation in patients with hereditary spastic paraplegia
Meyer T, Münch C, Völkel H, Booms P, Ludolph AC.J Neurol Neurosurg
Studies of the coding region of the neuronal glutamate transporter gene in amyotrophic lateral sclerosis
Meyer T, Lenk U, Küther G, Weindl A, Speer A, Ludolph AC.Ann
Publikationen mit Beteiligung der ALS-Forschungsgruppe an der Charité
Effect of High-Caloric Nutrition on Survival in Amyotrophic Lateral Sclerosis
Ludolph AC, Dorst J, Dreyhaupt J,
Prognostic factors in ALS: a comparison between Germany and China
Dorst J, Chen L, Rosenbohm A,
GABA B receptor encephalitis in a patient diagnosed with amyotrophic lateral sclerosis
Schumacher H, Meyer T, Prüss H.
Comprehensive analysis of the mutation spectrum in 301 German ALS families
Müller K, Brenner D, Weydt P,
Therapeutic decision in ALS patients: cross-cultural differences and clinical implications
Andersen PM, Kuzma-Kozakiewicz M, Keller J,
The metabolic and endocrine characteristics in spinal and bulbar muscular atrophy
Rosenbohm A, Hirsch S, Volk AE,
Hot-spot KIF5A mutations cause familial ALS
Brenner D, Yilmaz R, Müller K,
July 2017 ENCALS statement on edaravone
Al-Chalabi A, Andersen PM, Chandran S,
Genome-wide association analysis identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis
van Rheenen W, Shatunov A, Dekker
