Publikationen der ALS-Forschungsgruppe an der Charité
Point mutations of the p150 subunit of dynactin (DCTN1) gene in ALS
Münch C, Sedlmeier R, Meyer T, Homberg V, Sperfeld AD, Kurt A,
Chromosomal translocation t(18;21)(q23;q22.1) indicates novel susceptibility loci for frontotemporal dementia with ALS
Prudlo J, Alber B, Kalscheuer VM, Roemer K, Martin T, Dullinger J,
High rate of constitutional chromosomal rearrangements in apparently sporadic ALS
Meyer T, Alber B, Roemer K, Martin T, Kalscheuer VM, Göttert E,
The RNA of the glutamate transporter EAAT2 is variably spliced in amyotrophic lateral sclerosis and normal individuals
Meyer T, Fromm A, Münch C, Schwalenstöcker B, Fray AE, Ince PG,
The EAAT2 (GLT-1) gene in motor neuron disease: absence of mutations in amyotrophic lateral sclerosis and a point mutation in patients with hereditary spastic paraplegia
Meyer T, Münch C, Völkel H, Booms P, Ludolph AC. The EAAT2
Studies of the coding region of the neuronal glutamate transporter gene in amyotrophic lateral sclerosis
Meyer T, Lenk U, Küther G, Weindl A, Speer A, Ludolph AC.
Publikationen mit Beteiligung der ALS-Forschungsgruppe an der Charité
Identification of novel Angiogenin (ANG) gene missense variants in German patients with amyotrophic lateral sclerosis
Fernández-Santiago R, Hoenig S, Lichtner P,
Guidelines for the preclinical in vivo evaluation of pharmacological active drugs for ALS/MND: report on the 142nd ENMC international workshop
Ludolph AC, Bendotti C, Blaugrund E,
Familial ALS in Germany: origin of the R115G SOD1 mutation by a founder effect
Niemann S, Joos H, Meyer T,
The role of excitotoxicity in ALS – what is the evidence?
Ludolph AC, Meyer T, Riepe MW.J
Antiglutamate therapy of ALS – which is the next step?
Ludolph AC, Meyer T, Riepe MW.J
Molecular biology findings in amyotrophic lateral sclerosis
Bachus R, Claus A, Meyer T,
